While blocking FPR1 induces the inhibition of cellular processes important for tumor progression, it also results in the induction of factors such as VEGFA and angiopoietin 2, which are actively secreted by tumor cells into the tumor microenvironment where they not only constitute key growth factors for tumor development but also contribute to the formation of new vessels [8,17,30], thus suggesting that FPR1 could play a dual role as oncogene and onco-suppressor. The gene discussed is VEGFA; the disease is neoplasm.