In CS, also known as traumatic rhabdomyolysis, injured skeletal muscle cells collapse and release their contents, including myoglobin, creatine kinase, and electrolytes, into the circulation, leading to clinical complications such as myoglobinuria, acute kidney injury (AKI), electrolyte disorders, hypovolemic shock, and multiple-organ dysfunction (MODS) [2,3]. This evidence concerns the gene MB and acute kidney injury.