GPX4 and neoplasm: ATA noticeably down-regulated the protein expression levels of FDFT1 in tumor tissues of nude mice (Figure 8E,F), and it was found that the expression levels of the G1 phase (cyclin D1)-, apoptosis (pro-caspase3)-, and anti-ferroptosis (GPX4)-related proteins, as well as the autophagy receptor p62 were down-regulated, while those of the autophagy markers LC3 II/I were up-regulated in tumor tissues via ATA (Figure 8E,F), suggesting that ATA resulted in G0/G1 arrest and promoted apoptosis, ferroptosis, and autophagy in xenograft tumors, consistent with the in vitro experiment.