INHA and tuberculosis: Genetic analysis revealed (1) heterogeneity in the presence/absence of mutations to the Mtb genes associated with resistance to isoniazid (katG, inhA), rifampicin (rpoB), ethambutol (embB), fluoroquinolones (gyrA), capreomycin and aminoglycosides (rrs, eis) and (2) variation in the number of Mtb genes affected by these mutations among the patients regardless of the duration of intensive anti-TB chemotherapy before surgery (Table 1).