The heterogeneity of the results in the literature regarding the impact of anti-AMPA receptor autoantibodies in neurons, however, is not limited to anti-GluA3 antibodies, but also concerns the impact of other anti-AMPA receptor antibodies, specifically the anti-GluA1 and the anti-GluA2 receptor antibodies, which are overexpressed in the CSF and the serum of patients suffering from anti-AMPA receptor encephalitis [101,110]. This evidence concerns the gene GRIA1 and viral encephalitis.