Abnormalities in HDAC expression are often found in the most common types of oncological diseases, in particular, in various histological forms of non-small-cell lung cancer [333,334,335,336], prostate cancer [337], gastrointestinal malignancies [332,338,339,340,341,342,343], glioma [344], etc. Beyer et al. [345] found that the overexpression of HDAC3 mediates the growth of transformed cells in human acute myeloid leukemia by modulating the leukemia-related transcription factors β-catenin, the Wilms tumor suppressor gene (WT1), and the myelocytomatosis oncogene (MYC). Here, MYC is linked to non-small cell lung carcinoma.