Moreover, chemerin has been identified as a key factor in controlling lipid metabolism, particularly in the liver, by promoting lipid accumulation via lipogenic gene expression and triglyceride (TG) synthesis in hepatocytes, reducing their breakdown, and contributing to the development of non-alcoholic fatty liver disease (NAFLD) [32,33,34]. The gene discussed is RARRES2; the disease is metabolic dysfunction-associated steatotic liver disease.