We also found gender-wise differences of platelet indices and serum lipid profiles in ITP, which could also be found in healthy groups [22], presumably explained by (1) the differential distribution of lipids between genders, for example, higher Cho, HDL-C, ApoA1, NEFA and lower Hcy in female patients; (2) gender-different hormones, such as estradiol, which has been shown to affect thrombopoiesis [30]. Here, APOA1 is linked to autoimmune thrombocytopenic purpura.