Filbin et al. reported that the severity of the COVID-19 infections might shape the proteomic profile in plasma since COVID-19 patients with severe symptoms displayed higher expressions of proteins involved in the viral response and the IFN pathway including the proinflammatory cytokines such as CXCL10, CXCL11 as well as others involved into the response to vaccines, T cell function and innate immune activation [44]. The gene discussed is CXCL10; the disease is COVID-19.