To further demonstrate the role of the YAP/TAZ-TEAD signaling module in the regulation of TRAIL sensitivity in cancer cells, A549 cells expressing TEADi, an inhibitor of the interaction of YAP and TAZ with TEAD [44], were generated through infection with a tetracycline-inducible TEADi-GFP lentiviral vector (Figure 4C). This evidence concerns the gene TNFSF10 and infection.