Although autoantibodies for various antigens—such as cardiac myosin (Myhc), adenine nucleotide translocator (ANT), branched-chain α-ketoacid dehydrogenase kinase (BCKDk), sarcoplasmic/endoplasmic reticulum Ca2+ adenosine triphosphatase 2a (SERCA2a), β1 adrenergic receptor (β1AR), muscarinic M2 acetylcholine receptor, mitochondrial M7, and cardiac troponin I (cTNI)―have been detected in DCM patients [16,17,18,19,20,21,22,23,24,25], their role remains unclear, and isolated reports are available as to the role T cells play in DCM’s pathogenesis [26]. The gene discussed is BCKDK; the disease is familial dilated cardiomyopathy.