Numerous patients with acquired idiopathic (ai)‐TTP have a disintegrin‐like and metalloprotease with thrombospondin type 1 motifs 13 (ADAMTS13) inhibitor and reduced ADAMTS13 activity, which cleaves von Willebrand factor (vWF), resulting in unusually large vWF multimers binding to platelets and causing microvascular thrombi.2 This evidence concerns the gene VWF and thrombotic thrombocytopenic purpura.