MTOR and neoplasm: As the most important component of glutamine metabolism, glutamine metabolic enzymes can affect tumour cell proliferation and metastasis through a variety of pathways, including regulating dry properties through ROS/Wnt/β-catenin signalling, promoting cell proliferation and metastasis through interaction with miRNAs, regulating the expression of tumour EMT-related molecules, and promoting AMPK activation and mTOR signalling inhibition to affect tumour cell metabolism and metastasis [130].