WIPF1 and neoplasm: However, the cytoplasmic calcium increased by mitochondrial division in macrophages can eliminate the phase transformation of the Wiskott-Aldrich syndrome protein-Wiskott-Aldrich syndrome interaction protein (WIP) complex and phosphorylate WIP through protein kinase C-θ (PKC-θ), which is a vital link for tumour cells to escape from phagocytosis [120].