Of these target genes, FoxM1 is overexpressed in GSCs, and is required for GBM mesenchymal properties and chemoresistance, as we previously reported.[2b] We demonstrated that Rbfox2‐induced splicing of FoxM1 renders its phosphorylation by MEK1, and thus induces its nuclear translocation and stabilization. The gene discussed is FOXM1; the disease is glioblastoma.