Recent studies have reported that FBXO7 is a stress‐induced protein, and the amino acids T22, R378, and R498, which are frequently mutated in Parkinson's disease, are associated with FBXO7 stability in a proteasome‐dependent manner.[7, 32] Further, a chemotherapy‐induced protein, Pink1, has been shown to stabilize FBXO7.[33] Thus, it will be interesting to determine the role of those sites in mediating FBXO7 stabilization and upregulation during GBM MES transformation and acquired chemoresistance. Here, PINK1 is linked to Parkinson disease.