We document that wounding enhanced the number of transitional M1‐M2 macrophages (CD206+CD11c+; p = 0.0348) and M2 macrophages (CD206+; p = 0.0196), populations associated with tumor progression and immune suppression, and inhibited the number of M0 macrophages (CD206−CD11c−; p = 0.0436) (Figure 5b). Here, MRC1 is linked to neoplasm.