JUNB and cancer: Abundant evidence emphasizes the propensity of cancer cells to exploit the RNA epitranscriptome to escape immune surveillance through the regulation of glycolytic metabolism, adapt to nutrient‐deficient circumstances and maintain their growth and proliferation.[24] For instance, FTO‐mediated m6A modification regulates some TFs, such as c‐Jun and JunB, which subsequently dismantle the metabolic barrier, impeding T‐cell activation.