IL17A and COVID-19: Th17 cell populations are increased and activated in patients with COVID-19 that exhibit pulmonary symptoms.136 Furthermore, hyperinflammation and lung injury in such patients are correlated with increased Th17 cell responses,137 neutrophilia, and increased NETosis.138 In addition, IL-17 stimulation is partly driven by Candida colonization, and blunted type I/III IFN signaling is a common feature of severe COVID-19 infection.139