The IL-17 response prevents colonization of the nasopharynx by Streptococcus pneumoniae via neutrophil recruitment and pneumococcal eradication.117–119 However, IL-17 can also increase vulnerability to pulmonary Acinetobacter baumannii infection in mice by inhibiting neutrophil phagocytosis during the early stages of infection.120. The gene discussed is IL17A; the disease is infection.