TP53 and type 2 diabetes mellitus: GSEA suggested that “cell cycle”, “DNA replication”, “p53 signaling pathway” and “spliceosome” were remarkably enriched in ERGcluster B, “maturity onset diabetes of the young”, and “neuroactive ligand receptor interaction” were remarkably enriched in ERGcluster A (Figure 3F), which was consistent with the GSVA result.