Yu's research showed82 that by inhibiting the PI3K/Akt pathway, GC cell proliferation and apoptosis can be effectively inhibited; in addition, Liu's team found that EA inhibited the growth and proliferation of non‐small cell lung cancer cells by downregulating the PI3K/Akt pathway83 and induced apoptosis by regulating the expression of apoptosis‐related proteins Bax, Bcl‐2 and caspase‐3 through this pathway. Here, AKT1 is linked to lung cancer.