On the contrary, levels of Cldn1,Cldn4,Cldn6,Cldn9,Cgn,F11r, and Cdh1, associated with exacerbated inflammation, perturbation of the TJ assembly [27, 40], and even with mesenchymal transformation or tumour progression [41, 42], were overexpressed. This evidence concerns the gene CLDN1 and neoplasm.