Our results showed that PYJ remarkably descended protein expressions of ICAM-1, VCAM-1, MCP-1, and COX-2 in uteri of PID rats (Figure 5), especially in the sites of inflammatory cells infiltration, suggesting PYJ attenuates chronic inflammation and pelvic adhesion of PID by suppressing these pro-inflammatory mediators. The gene discussed is CCL2; the disease is pelvic inflammatory disease.