Other relevant pathways included neuron remodeling, synapse pruning, regulation of tau-protein kinase activity, regulation of behavioral fear response for human AD MG DEGs, and negative regulation of amyloid fibril formation, high-density lipoprotein particle clearance, negative regulation of synaptic potentiation, and regulation of T/B cell immunity for mouse AD MG DEGs. Here, WEE1 is linked to Alzheimer disease.