For example, Wang et al. (2015) suggested that RILP as a therapeutic target against breast cancer, and that hyper-expression of RILP suppressed the proliferation, migration, and invasion of breast cancer by inhibiting the interaction of Ras-like proto-oncogene A (RalA) and Ral guanine nucleotide dissociation stimulator (RalGDS). The gene discussed is RALA; the disease is breast carcinoma.