Little is known about the function of INHBE in the context of endothelial cells, but the significant association of this protein with CM and neurological complications— together with lack of association with SM in general, other nonneurological complications (acidosis, anemia, or jaundice), and P. falciparum biomass — suggests that it may be specific to brain endothelial damage, to some extent. Here, INHBE is linked to Acidosis.