Co‐culturing with the Arf1‐ablated tumor cells led to significantly increased expression of Ifna, Ifnb, and Cxcl10 in the DCs (Figure 1g), while this effect was abolished after knocking down Cgas or Sting1 genes in the DCs (Figure 1g and Figure S4a, Supporting Information), suggesting that the co‐cultured DCs produced type I IFNs and Cxcl10 in a cGAS‐STING signaling dependent manner. The gene discussed is CGAS; the disease is neoplasm.