eBL is characterized by EBV infection and translocation and dysregulation of the proto-oncogene MYC. Studies have shown that the dysregulated expression of activation-induced cytidine deaminase (AID) can lead to the translocation of MYC in cells infected with latent EBV, thereby promoting the occurrence and development of BL [115]. The gene discussed is AICDA; the disease is Epstein-Barr virus infection.