ADARB2 and glioblastoma: In a recent publication comparing A-to-I editing in U87 glioblastoma cells (which endogenously express low levels of ADAR3) to editing in U87 cells with higher levels of ADAR3 exogenously introduced, Kurup et al. determined that transcripts exhibiting reduced A-to-I editing in glioblastoma are more likely to be bound to ADAR3.