Testing another tumor model, we found that the administration of allogeneic C57BL/6‐derived NK cells together with anti‐NKG2A/Qa‐1b mAbs led to control of tumor progression and enhanced survival of BALB/c mice engrafted with the CT26 colon cancer cells to form established subcutenous tumors (Fig 2D–F). The gene discussed is KLRC1; the disease is neoplasm.