Furthermore, in utero ethanol exposure was recently shown to disrupt cIN migration in a mouse model of fetal alcohol spectrum disorder (FASD; Skorput et al., 2015; Lee et al., 2022), suggesting that environmental factors, together with perturbations of intrinsic molecular programs, both play critical roles in cIN development, relevant to a range of neurodevelopmental disorders. Here, PDXP is linked to neurodevelopmental disorder.