Although chiauranib is a multi-target inhibitor against aurora B kinase, VEGFR, colony-stimulating factor 1 receptor, platelet-derived growth factor receptor in tumor angiogenesis [60], the results of MSCs and VEGFA co-culture with KGN and GCs revealed that chiauranib targeted VEGFR2 on the cell membrane, and suggested that paracrine VEGFA of hUC-MSCs mitigated the CTX-induced excessive autophagy. The gene discussed is AURKB; the disease is neoplasm.