IL‐1β, a pro‐inflammatory factor that promotes tissue damage during viral infection,10, 11 was also increased in the CDVC group (25.8 pg/mL, range 0–264.7 pg/mL), and its level was significantly higher than the perished‐ and the recovered‐ groups, highlighting it may contribute to the clinical phenotypes observed (Figure 1A, Table 1). This evidence concerns the gene IL1B and viral infectious disease.