TLR2 and colitis: Apart from Lactobacillus reuteri, Lactobacillus rhamnosus was also competent in interfering with the relative abundance of immune cells since it was proved to decrease the Th17/Treg ratio through the JAK-STAT signaling pathway with the presence of toll-like receptor 2 (TLR2) in the colon of mice with DSS-induced colitis [18].