Another study by Christian W. Keller et al. compared the MOGAD group (n = 109) with the MS group (n = 34), NMOSD group (n = 13), and healthy controls (n = 16), indicating that complement indicators C3a, C5a, SC5b9, Ba, and Bb had the highest concentrations in MOGAD, suggesting that the activated complement system plays a significant role in the pathogenesis of MOGAD (27). The gene discussed is C3; the disease is myeloid sarcoma.