B3GNT9 and juvenile Huntington disease: The results showed ten significant Kyoto Encyclopedia of Genes and Genomes (KEGG) pathways in the β3GNT9 high expression phenotype, including cardiac muscle contraction, extracellular matrix receptor interaction, glycosaminoglycan biosynthesis chondroitin sulfate, glycosaminoglycan biosynthesis heparan sulfate, glycosaminoglycan biosynthesis keratan sulfate, glycosaminoglycan degradation, Huntington’s disease, oxidative phosphorylation, Parkinson’s disease, riboflavin metabolism (Figure 5A).