A previous study from Spain reported causal genetic variants in 73% of 52 transplant patients with familial DCM, including 25% with a founder mutation in the emerin (EMD) gene.25 Another study employed gene panel testing of peripheral blood from 31 heart transplant patients with nonischemic cardiomyopathy and identified pathogenic and likely pathogenic variants in 38.7%.26 This evidence concerns the gene EMD and nonischemic cardiomyopathy.