Finally, in vitro exposure of bronchial epithelial cells (IB3-1) to the SARS-CoV-2 spike protein and sulforaphane display reduced gene expression of the inflammatory interleukins IL-6 and IL-8, which are key components of the cytokine storm and potentially resultant acute respiratory distress syndrome (ARDS) [34]. The gene discussed is CXCL8; the disease is acute respiratory distress syndrome.