Analyzing the correlation between MCP1 and PUFAs, in CKD patients, we observed an increasing direct correlation of MCP1 with some n-6s (DGLA, Osbond, ARA, GLA) and a negative correlation with n-3 between stages, while in KTx patients, no correlation was observed following the fact that the absence of increased MCP1 could avoid its influence on the PUFA balance in favor of the pro-inflammatory one. Here, CCL2 is linked to chronic kidney disease.