We have also identified genes being increasingly expressed by both sources of LPS, such as UNC5CL, an inducer of a pro-inflammatory signaling cascade [74]; PTGER4, a prostaglandin E2 receptor expressed in oral squamous cell carcinomas [75] including HSC3 [76]; killin, a p53-regulated nuclear inhibitor of DNA synthesis [77]; and PPP1R3F, the protein phosphatase 1 regulatory subunit 3F. The gene discussed is KLLN; the disease is oral cavity squamous cell carcinoma.