TNFRSF12A and colorectal carcinoma: To ask if depleted Gln/Glutaminolysis and TCA byproducts are key effectors of Tnfrsf12a expression, CRC cells were deprived of Gln but supplemented with either DM-aKG, succinate (SUC), or antioxidant N-acetyl-L-cysteine (NAC).