We selected every significant virulence determinant, like genes and their genotypes or status (e.g., presence of cagA/cagPAI, sabB, complete tfs4, tfs4b type, ceuE, hopD, or frpB2 gene; status of vacA s1i1m1 allele, hopQ-I allele, sabB “on” and oipA “on”) linked to GC, in order to determine the relationships between each other, and which virulence factors had the most impact on gastric diseases. This evidence concerns the gene S100A8 and stomach disorder.