Comparative increases in neurogenesis (F(3,28) = 147.1, p < 0.001) were also observed in both juveniles (26.7 ± 1.8%) and adults (27.2 ± 1.8%, p = 0.9924) in the stroke-injured hemisphere, evidenced by increased colocalization of BrdU+/DCX+ cells (Figure 1A,B,E, white arrows), demonstrating an increased proliferation and migration of newborn neurons to the site of injury, compared with an absence of neurogenesis (0%, p < 0.0001 in both age groups) in the uninjured hemisphere. This evidence concerns the gene DCX and stroke disorder.