Accordingly, the MSC-based suppression of NLRP3/IL-1β/IL-18-dependent activation of eye-infiltrated NK, NKT cells, Th1, and Th17 lymphocytes crucially contributes to the attenuation of T and NK/NKT cell-driven inflammatory eye diseases, including DED, herpetic stromal keratitis, uveitis, and Graves’ disease [4,8,21]. This evidence concerns the gene IL1B and uveitis.