These abnormal toxic proteins (Aβ) in AD [44], α-synuclein in PD [47,48], and SOD1 in ALS [46], abnormally present Fe2+ and Cu+ ligands for inappropriate chemical reactions with H2O2 called Fenton and Haber–Weiss reactions, respectively (Figure 1) [9]. This evidence concerns the gene SOD1 and amyotrophic lateral sclerosis.