The enhanced cellular metabolism, mitochondrial polarization, and ER stress in the YWHAG regulome are suggestive of elevated oxidative stress in cancer cells undergoing EMT.[18] We measured higher mean intracellular reactive oxygen species (ROS) in MKN74 cells during DMOG‐ and TGF‐β1‐induced EMT than in untreated MKN74 cells (Figure 3A; Figure S9A, Supporting Information). Here, TGFB1 is linked to cancer.