In addition and supporting, suppression of IL‐7 receptor signalling and additive genetic loss of Xbp1 resulted in increased phosphorylation of P38‐T180/Y182 and JNK‐T183/Y185 in pre‐B NRASG12D ALL cells, leading to cell death mediated by activation of stress response kinases (Figure S2B). The gene discussed is XBP1; the disease is acute lymphoblastic leukemia.