using a murine NAFLD model divided into control and NAFLD groups; they observed that a plasma decrease in Unacylated Ghr/Acylated Ghr ratio in combination with the hypothalamic over-expression of Acylated Ghr and its receptor could be associated with NAFLD due to their positive correlations (R2 = 0.56 – 0.85, p <0.05) with homeostatic model assessment insulin resistance (HOMA-IR) (20). The gene discussed is GHR; the disease is metabolic dysfunction-associated steatotic liver disease.