The genetic profile of liver tissue derived from patients with NAFLD, reported in the Gene Expression Omnibus database, shows an overexpression of lipogenic enzymes, such as ELOVL6, SCD1, both usually absent in normal hepatocytes, and associated with a decreased hepatic steatosis and adiposity [47], as well as proteins like TNF-α, MLKL and RIP-1, which have been linked to an augmented uptake of PA [31]. Here, MLKL is linked to Hepatic steatosis.