MET and myotonic dystrophy type 1: Using these data, we selected eight direct targets of miR-23b that were both significantly repressed or unchanged (Figure S4A) in a CNT vs. DM1 comparison, and were significantly upregulated comparing control and DM1 muscle cells treated with the corresponding antagomiR (CHUK, SRC, NOTCH2, TAB2, SIRT1, HMGB2, MET, and PLAU).