Notably, the chemokine CCL2 recruits myeloid cells to the tumor stroma and promotes immune evasion.[40, 41] To further investigate the interaction between CAFs and myeloid cell subpopulations, we performed cellcell communication analysis using CellCall and found that the interaction between ICAM1+ CAFs and THBS1+ monocytes was enriched in the adherens junction and chemokine signaling pathway (Figure 6C,D). This evidence concerns the gene ICAM1 and neoplasm.