CCL2 and cancer: Furthermore, CCL2 and CCL11 were highly expressed in the CAF subclusters associated with recurrence in the ovarian cancer single‐cell dataset (Figure 6G).[32] Taken together, these findings suggest that the ICAM1+ iCAFs may recruit MDSCs (THBS1+ monocytes) to mediate immune suppression in recurrent cancers via the CCL2‐CCR2 signaling axis.