Importantly, the expression of some genes known to be involved in the most important forms of SNHL [135, 136], such as NEFL (a neuron-specific intermediate filament essential for the radial growth of axons), TPRN (taper-in), HMOX1 (heme-oxygenase1), OTOGL (otogelin-like protein), GJB2 (Cx32) and REST (a DNA-binding protein that complexes the histone deacetylases), appeared unregulated following the chronic EMF exposure (Fig. 4). This evidence concerns the gene OTOGL and sensorineural hearing loss disorder.